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From a pathophysiological perspective, pancreatic cancer is a disease with a prolonged latency, typically requiring years of subclinical progression before clinical manifestation [37]

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Assessing growth and response to therapy in murine tumor models

When cells are exposed to oxidative and/or electrophilic stress, KEAP1 thiol residues are directly modified, which inactivates the ubiquitin E3 ligase activity of KEAP1-CUL3 complex, leading to stabilization of NRF2