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It is not only closely linked to the systemic complications of diabetes but also involves specific pathological changes in myocardial metabolism and oxidative stress ( Figure 1 )

GHK-Cu activates fibroblasts, the cells responsible for collagen and elastin synthesis, and triggers extracellular matrix remodeling through metalloproteinase regulation

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It works by stimulating lipolysis (the breakdown of fat) while inhibiting lipogenesis (the formation of new fat)