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For example, in G6PD-deficient individuals, residual hepatic enzyme activity can prove insufficient in stress states where increased G6PD activity is required, as in neonates with comorbid G6PD deficiency and Gilberts syndrome, who are at increased risk for hyperbilirubinemia (96, 97)

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MOTS-c similarly costs $100 to $200 research-grade

Mutations in the nuclear bile acid receptor FXR cause progressive familial intrahepatic cholestasis

Or that certain conditions and diets can cause a vitamin deficiency or affect how nutrients are digested and metabolized