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However, despite the fact the GPER is known to stimulate the cAMP-PKA pathway (Prossnitz and Barton, 2014), H89 did not affect the protection by G1 (data not shown)
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These findings show that diallyl disulphide can attenuate cyclophosphamide mediated by cystitis through blocking inflammation mediated by NF-B and MAPKs pathways and the related antioxidative effect
Features include thickening of the skin, skin ulcers and stiff joints
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