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Furthermore, Oxford Academic publications [4] report that disruption of MC4 receptor function produces hyperphagia and obesity phenotypes, further reinforcing receptor-specific mechanisms involved in appetite regulation

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Furthermore, the option to continue or initiate new AD medication during the trial, including the amyloid lowering monoclonal antibodies, was not a randomization stratification factor and may therefore act as a confounding factor if their use across treatment arms is unequal

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