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Similarly, exenatide-4 inhibits hepatocarcinogenesis independent of obesity and MASH resolution, acting through the cAMP-PKA-EGFR-STAT3 axis and mTOR modulation to suppress proliferation and reduce HCC multiplicity (Zhou et al., 2017)

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(57) first reported dramatic improvement in PsO symptoms in a 73-year-old patient who had previously been unresponsive to biologic therapy