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However, the precise mechanisms of ferroptosis in CHD have not been fully elucidated, necessitating further in-depth mechanistic investigations and the development of more effective and safer clinical intervention strategies to ultimately improve patient outcomes

Increased levels of ROS and lipid peroxidation products and/or decreased levels of antioxidant enzymes, such as superoxide dismutase (SOD) and catalase, and antioxidant compounds, such as glutathione, have been reported in both NAFLD/NASH patients as well as a variety of animal models of NAFLD 9,10,11,12,13,14

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