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After prolonged physical exertion, glutathione neutralizes reactive oxygen species (ROS) produced during the Krebs cycle, effectively preventing the lipid peroxidation of mitochondrial membranes and allowing them to recover and maintain efficient ATP production without suffering from premature fatigue or permanent oxidative damage.

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this activates the tubulo-glomerular feedback responsible for afferent arteriole dilation and efferent arteriole constriction and, ultimately, leads to glomerular hyperfiltration and hypertension

The low urea level, obtained at the end of 10 d of this study, is consistent with the findings of Severin et al