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In microglial BV2 cells semaglutide dose-dependently inhibited apoptosis induced by -amyloid (1-42) and promoted the transformation of microglia from M1 to M2, thereby exerting anti-inflammatory and neuroprotective effects [50]

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For example, a recent, small study of women with PCOS and obesity found that six months of low-dose semaglutide normalized menstrual cycles and significantly lowered body mass index (BMI) in 80% of participants