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Together, these observations support a model in which pathological consequences of altered melatonin rhythms in asthma may arise from a temporal mismatch between melatonin availability and NRF2-driven redox programs in key immune cell populations such as ILC2s
In: Chapter APsDA-W
In case of Drosophila , neuronal redox balance was altered by driving CatalaseRNAi or PHGpxRNAi, along with roGFP by elav-Gal4 to deplete the respective antioxidant enzymes and express the redox biosensor
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