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doi: 10.1080/15548627.2024.2410621 157 AiSTaoJWangYZhouJLiuWLiXet al
Postmortem analysis has revealed that insulin resistance (IR) also occur in AD patients brain with significantly decreased expression of the insulin receptor, concomitant with the disease progression (66, 67) and indicated that the defects in insulin signaling are associated with AD pathogenesis (68)
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GLP-1based therapies are not positioned as replacements for established treatments but as complementary agents that may enhance overall therapeutic efficacy
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