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In particular, LEAP2 (liver-expressed antimicrobial peptide 2), an endogenous antagonist of the ghrelin receptor GHSR, has emerged as a potential target for mitigating the weight regain often observed after discontinuation of GLP-1-based therapies [22,23]
Recent research employing radiolabeled MMP PET has revealed that heightened MMP activity is localized around active lesions, which are linked to leukocyte infiltration [262]
The peptides are then combined and processed through a freeze-drying step, which removes solvent and water while preserving peptide structure and biological characteristics
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