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Supplemental ascorbate diminishes DNA damage yet depletes glutathione and increases acute liver failure in a mouse model of hepatic antioxidant system disruption

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GHK-Cu is investigated in preclinical and dermatological research for its effects on collagen and glycosaminoglycan synthesis, fibroblast activation, angiogenesis, copper-dependent enzyme function, antioxidant pathways, and DNA repair gene expression

The emission from intracellular region covers the green and yellow channels (Figure 7AC)

Statements Author contributions FQ: Methodology, Writing original draft