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Postconditioning with irisin attenuates lung Ischemia/Reperfusion injury by suppressing ferroptosis via induction of the Nrf2/HO-1 signal axis

Higher mitochondrial metabolism in the neurons result in generation of Complex I dependent hydrogen peroxide and neuronal membranes, rich in polyunsaturated fatty acids, are particularly vulnerable to oxidative stress due to their possession of unsaturated double bonds

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