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In hepatic IRI rats, pretreatment with SFN could alleviate liver injury by inhibiting oxidative stress and improving antioxidative activity as well as mitochondrial function in liver tissues, which was accompanied with the activation of Nrf2 pathway (Zhao et al., 2010
TB-500's primary mechanism is upregulating actin, a protein essential for cell structure, movement, and repair
On the one hand, GSH binds to reactive metabolites, preventing them from interacting with cellular macromolecules
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