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Evidence from receptor blockade and GLP-1R gene knockdown experiments confirmed that the spinal cord is a primary site mediating exenatide-induced antinociception [24]

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It is added because it addresses two specific and significant clinical problems that arise from GLP-1 receptor agonist therapy: nausea at treatment initiation and progressive B12 depletion over the course of long-term use

Association between adverse maternal and embryo-fetal effects in norfloxacin-treated and food-deprived rabbits