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In a research context, this solution serves as a critical supplement in cell culture media to support normal cell growth and metabolism

The vitamin is then transferred to TC2, which serves as the circulating plasma transporter, hereditary defects in the production of TC2 and its receptor may produce functional deficiencies in B 12 and infantile megaloblastic anemia, and abnormal B 12 related biochemistry, even in some cases with normal blood B 12 levels

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However, AOD 9604 faces a significant regulatory challenge

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