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Modulation of diabetes-induced oxidative stress, apoptosis, and Ca 2 + sup 2 + entry through TRPM2 and TRPV1 channels in dorsal root ganglion and hippocampus of diabetic rats by melatonin and selenium
CJC-1295 with DAC progressed through Phase 2 trials but did not advance to a marketed therapeutic
The study also demonstrated the protective effect of exenatide on islet-cell function during GCs therapy, providing early evidence for using GLP-1 RAs to counteract the diabetogenic effects of GCs (34)
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