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Pathological stimuli-including inflammatory cytokines and oxidative stress-trigger IKK-mediated IB degradation, enabling NF-B nuclear translocation and transcription of pro-inflammatory cytokines and atrophy-related genes (Scalabrin et al., 2020)

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However, GIPR agonism can potentially decrease the nauseous effects of GLP1 agonism by inhibiting area postrema inhibitory neurons

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