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In studies with mouse models of NAFLD and acute kidney injury (AKI), treatment with ACMSD inhibitors increased the transcription of mitochondrial and -oxidation genes, as well as restoring NAD + levels in the kidney and liver 235
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PT-141 was derived from Melanotan II by removing the cyclic lactam bridge to produce a linear metabolite with reduced MC1R affinity while retaining MC3R and MC4R agonism, effectively separating melanogenic activity from central nervous system-mediated behavioral effects
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