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The design of drug targets for ASCT2 typically follows a substrate analogue approach, wherein substrate analogues function as competitive inhibitors, thereby reducing the uptake of essential amino acids by cancer cells
This duality makes glutathione a focal point in efforts to fine-tune redox modulation during cancer prevention versus cancer treatment
Pro-inflammatory cytokines like TNF-alpha, IL-1beta, and IL-6 damage surviving nerve fibers, interfere with Schwann cell repair functions, and create a hostile environment for axonal regrowth
H.et al (2020)
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