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HMGB1 translocation from nucleus to the cytoplasm of renal tubular cells induces ferroptosis by binding ACSL4 after renal I/R injury, and inhibition the nuclear cytoplasmic translocation of HMGB1 could inhibit ferroptosis and renal I/R injury

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However, when GPX4 activity or GSH availability is diminished, lipid peroxide accumulation increases, facilitating ferroptosis initiation [21, 165]