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It was previously reported that targeted FUS-induced BBB opening facilitates CNS penetration of systemically administered anti-A antibodies to targeted brain regions in the TgCRND8 and APP23 mouse model of AD, therefore reducing plaque load 5 , therefore confirms the feasibility of this strategy in promoting behavioral and memory recovery in a transgenic animal model 6,7

The FDA has not approved them for human use in the US

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It facilitates cell migration to injury sites, promotes cell differentiation, supports actin-mediated tissue remodeling, and reduces scar tissue formation

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