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The ability of lipids to induce CCK release and activate VANs to lower food intake may be due to chylomicron formation and subsequent activation of sensory machinery on the basolateral side of EECs, as mentioned previously
Behrens SC, Lenhard K, Junne F, Ziser K, Lange J, Zipfel S, et al
The early experience on GLP-1 compounds provides useful parallels, though the retatrutide experience includes the unique glucagon elements
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