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Concurrently, high glucose suppressed the tumor suppressor TIF1, further amplifying Smad-mediated transcription and elevating the expression of pro-oncogenic genes, including the epithelialmesenchymal transition markers N-cadherin and vimentin, thereby collectively fostering CRC progression
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4144 This activation enhances the activity of POMC neurons, which induce satiety, while suppressing NPY neurons that drive hunger
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