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Finally, they reflect pathways likely additional to those involved in the angiogenesis (Vukojevic et al., 2018), recently associated in particular with the increased expression, internalization of VEGFR2, and the activation of the VEGFR2-Akt-eNOS signaling pathway (Hsieh et al., 2017) (see Genes Expression as Support ), given the rapid presentation of all of these effects, which occurred before the process of angiogenesis could be initiated (Sikiric et al., 1993
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Synthesis and biological properties of -glutamyl-dermorphin, a prodrug
Increasing the number of EBV-dormant cells would also increase the evasion mechanisms of these cells by decreasing the activation and cytotoxic capacity of EBNA-1-specific CD4 T cells through the release of IL-10 (increased humoral Th2 response), chemotaxis of regulatory T lymphocytes (Treg) due to increased CCL20 expression in EBNA-1-mediated EBV latency cells, and release of EBV miRNA contained in exosomes, which could suppress the expression of target genes in the viral or host genome to maintain latent EBV infection [206,207,208]
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