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Acknowledgements The authors are supported by US NIH grants R01-DK55758, R01-DK099110, R01-DK127274, R01-DK143576 and R01-DK131537 and P01-AG051459 to P.E.S

215 Although both ER-alpha and ER-beta are important for ovarian development and function and cardiovascular protection, ER-alpha is the more prominent mediator of estrogens effects on metabolism and the breasts, uterus, and bones, whereas ER-beta is more involved in mediating nervous system and immune effects of estrogen, and counteracts pro-proliferation signaling by ER-alpha in the breasts and uterus

The Precision Peptide Genetic Test reveals predispositions in pathways including FTO rs9939609, MC4R rs17782313, GIPR rs1800437markers that may provide broader context about metabolic regulation and tissue remodeling capacity

showed that exposure to nickel chloride in vitro induced lytic cell death and eryptosis, which is mediated by Ca 2+ influx and p38 MAPK signaling