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Main Research Findings 1) Administration of GHRP-6 was shown to prevent doxorubicin-induced myocardial damages by activating various prosurvival mechanisms
The core mechanism of action of IGF-1 LR3 IGF-1 LR3 activates downstream signaling pathways (such as the AKT/mTOR pathway) by binding to the IGF-1 receptor (IGF1R) on the cell surface, regulating cell proliferation, differentiation, migration, and metabolism
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