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Restoring mitochondrial biogenesis with metformin attenuates -GP-induced phenotypic transformation of VSMCs into an osteogenic phenotype via inhibition of PDK4/oxidative stress-mediated apoptosis

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When medication potency decreases, patients with type 2 diabetes may experience inadequate glycemic control, with blood glucose levels rising above target ranges

GPx4 is also a major structural protein of the capsule embedding mature sperm mitochondrial helix involved in sperm motility

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