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Accumulating evidence indicates that elevated levels of IR and circulating FFAsparticularly palmitatein T2DM contribute to -cell ERS through pathways including inositol-requiring enzyme 1 (IRE1)/X-box binding protein 1 (XBP1), protein kinase R-like endoplasmic reticulum kinase (PERK)/stimulates eukaryotic translation initiation factor 2 subunit- (eIF2), activating transcription factor-6 (ATF6), and thyroid adenoma associated gene (THADA) [70]

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