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The entry of cisplatin into the inner ear leads to the formation of inner ear cellular damage products (DAMPs), which, together with cisplatin, bind to toll-like receptors (TLRs) (Babolmorad et al., 2021) and activate downstream signaling, producing transcription factors (e.g., nuclear factor kappa B (NF-B), activator protein-1 (AP-1), and interferon regulatory factor 3 (IRF3)), which in turn regulate the expression and secretion of proinflammatory signaling molecules

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After 3 months of semaglutide, the percentage of patients with EE decreased from 72.5 to 11.5%, the percentage of patients with external eating decreased from 27.5 to 10.1%, the percentage of patients with craving decreased from 49.3 to 21.7%, and the percentage of patients with savory craving decreased from 53.6 to 14.5% [59]

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