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The Arg3 to Leu3 substitution disrupts a key contact residue in the IGFBP binding interface, and the N-terminal extension further sterically interferes with binding pocket access
Journal of Clinical Investigation , 96 , 710720
BPC-157's preclinical record in animal models is genuinely compelling the healing mechanisms proposed are biologically plausible and well-described [1]
Rheumatoid arthritis affects the joints
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