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The glucagon-like peptide 1 analogue exendin-4 attenuates the nicotine-induced locomotor stimulation, accumbal dopamine release, conditioned place preference as well as the expression of locomotor sensitization in mice

1c), which clearly labelled cells in the mitral cell layer (MCL) and GCL of regular diet (RD) mice (Fig

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By activating both phases concurrently, the blend compresses the healing timeline

Because MOTS-c promotes fat utilization, which may underly its effect on metabolic flexibility (Figs