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Neuroinflammation disrupts the neuronal microenvironment, interfering with drug action and accelerating the aggregation of alpha-synuclein 73
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The researchers commented that the peptide suppressed caspase-dependent apoptosis that increases during aging of cell cultures. By potentially restraining Caspase-3 activity, Epithalon might support cellular longevity and decrease apoptosis, enhancing its regenerative capabilities
As the name of this condition suggests, we know that inflammation is involved, but a 2020 study found that levels of glutathione were low specifically in the section of the colon where inflammation was present
