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AKT also phosphorylates the pro-apoptotic and anti-proliferative transcription factor FOXO1, which leads to its exclusion from the nucleus and cytosolic degradation, thereby connecting tumor cell metabolism with cell cycle progression and survival
Nutrient support Focused nutrient support (e.g., B12, B-complex)
GHK-Cu upregulates several tumor suppressor genes (caspases, growth arrest and DNA damage-inducible genes) and downregulates genes associated with metastasis, invasion, and angiogenesis
In rodent studies, these medications caused thyroid tumors including thyroid cancer
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