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These findings suggest that the nootropic compound elicits neuroprotective effects on nerve cells in the brain damaged by inflammatory factors [1]

The mechanism underlying these gastrointestinal cytoprotection-associated findings has been attributed in published research to NO-related maintenance of mucosal microvascular function and preservation of endothelial integrity, rather than to simple acid suppression

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BPC-157 research focuses primarily on animal models and in vitro studies, with limited human clinical trials