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10.1590/s1678-9946201961004 26 GreenG
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DSIP-CBBBP have inhibitory functions on the symptoms
Glucose-induced CRL4(COP1)-p53 axis amplifies glycometabolism to drive tumorigenesis
Similarly, the released thiol compounds from glutathione upon entering the cell might react with the thiol groups on other proteins such as Kelch-like ECH-associated protein 1, which may enhance translocation of nuclear factors (erythroid-derived 2)-like 2 (NRF2) into the nuclei and expression of antioxidant enzymes
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