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These exposures act via a mechanism involving hormonal dysregulation, oxidative stress buildup, DNA damage, and epigenetic alterations

Its primary mechanism is angiogenesis: BPC-157 helps trigger new blood vessel growth in injured tissue, which is what allows it to support repair in tendons, ligaments, the gut lining, and joints

GHK-Cu, by contrast, has only preclinical (laboratory and animal) evidence and has not been validated in large-scale human trials

Thus, oxidative phosphorylation in mitochondria can generate large numbers of oxygen radicals in the nervous system

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