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demonstrated that oxidative stress and H 2 O 2 treatment led to a marked increase in senescence-associated -galactosidase activity but only to minimal apoptotic cell death in CSCs, suggesting that ROS triggered the induction of senescence and exhibited therapeutic potential in the eradication of drug-resistant CSCs [8]
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In this blog, well look at how each treatment works, the key differences between them, and how to know which option is best suited to you
doi: 10.1002/art.43029 113 Jasiecka-MikoajczykASochaP
