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Although additional studies are required to clarify the molecular mechanisms responsible for hyperhomocysteinemia-induced vascular disorders, there is evidence that the direct cytotoxicity of markedly elevated homocysteine and/or of its oxidized by-products (homocysteine thiolactone, homocysteine sulfinic acid, and homocysteic acid) damages endothelial cells (Dudman et al, 1991

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[5] As all of these symptoms are all too common in Hashimotos, I designed a Liver Support Protocol to support the methylation pathways, back in 2015

Autophagy/mitophagy in airway diseases: impact of oxidative stress on epithelial cells