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& Zhao, C
It was demonstrated that glutaredoxin (Grx1), a cytosolic oxidoreductase, can efficiently deglutathionylate eNOS in the presence of GSH, i.e., when the cellular redox status is favorable [254]
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Numerous preclinical studies with pharmacological Nrf2 activators (e.g., sulforaphane, naringenin, curcumin) have demonstrated significant cardioprotection in MIRI models, attenuating injury, reducing infarct size, and decreasing oxidative stress, inflammation, and cell death (Wang R
doi: 10.1590/fst.24219 79
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