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Pathol.: Mechanisms Dis

60 In this bioinformatics-assisted review, a plausible molecular pathway is suggested whereby ROS-induced activation of Cdc42, in conjunction with Rac1 protein, initiates inflammatory signaling pathways, particularly RAC-alpha serine/threonine-protein kinase (Akt), and serine/threonine-protein kinase PAK 1/mitogen-activated protein kinases (MAPKs) such as ERK1/2, MAPK 8 (JNK), and p38 MAPK

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