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CIT functions as a precursor to L-arginine, thereby facilitating the synthesis of NO through the activation of eNOS, which in turn enhances endothelial functionality, vasodilation, and the perfusion of skeletal muscle (74)

Clinical data supports many of these benefits

The protective effects of probiotics are largely attributed to their capacity to suppress urease-producing, pro-inflammatory pathogenic bacteria, thereby reducing the risk of HE development (499)

Co-immunoprecipitation studies demonstrated that the peptide reduces eNOS/caveolin-1 binding by approximately 50%, effectively doubling the available eNOS for nitric oxide production

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