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Small molecule GLP-1 agonists and antagonists (such as those in development at Structure Therapeutics) may facilitate study of endogenous GLPs activity and increase clinical uptake in T2D and obesity patients

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Under physiological conditions, ROS are generated in the heart mainly from the mitochondrial electron transport chain, nitric oxide synthases (NOS), NADPH oxidases (NOX), and xanthine oxidase (XO) in small quantities [7, 8]

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