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Egr, Nos, Srf, Vegfr, Akt1, Plc, and Kras ) provide a likely special point to explain how the dysfunction and its counteraction is causal to, or result of (Vukojevic et al., 2018) (see Genes Expression as Support )
As well, KYNA-induced inhibition of 7nAChRs reduces presynaptic glutamate release, thereby indirectly dampening AMPAR activation, a mechanism that may protect neurons from glutamate-driven excitotoxicity under pathological conditions [82]