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Transforming growth factor-beta activation of phosphatidylinositol 3-kinase is independent of Smad2 and Smad3 and regulates fibroblast responses via p21-activated kinase-2

Ghati et al., 2021

We supplemented the crystal structure with site-directed mutagenesis to link the structural information of the isolated extracellular domain with the binding properties of the full-length receptor

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Perhaps most remarkably, data from the Connectivity Map (CMap) suggests GHK-Cu can reset the expression of thousands of genes, effectively shifting the bodys cellular reprogramming back toward a state of repair and regeneration