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In recombinant cell systems, it activates GIPR with potency similar to native GIP and GLP-1R with reduced but pharmacologically meaningful potency relative to native GLP-1 a biased-agonism profile researchers have termed twincretin pharmacology
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It is not a treatment guide, dosing guide or human-use instruction
These minor effects typically diminish with continued use as the body adapts to glutathione therapy
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