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The interactions with the oxidative stress are resumed in figure 2
F.VallerandD.YouwakimJ.GratuzeM.PetryF
For individuals with metabolic dysfunction-associated steatotic liver disease, formerly called non-alcoholic fatty liver disease, that difference could mean the gap between disease progression and reversal
COX-2 may contribute to the formation of chloasma by activating tyrosinase and melanogenesis-related molecules, therefore promoting melanogenesis (Rodriguez-Arambula et al., 2015)
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