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It binds to GHRH receptors on pituitary somatotroph cells, stimulating the synthesis and secretion of endogenous growth hormone in a natural pulsatile pattern (1)

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In summary, our findings demonstrate that METTL3-mediated m 6 A modification acts as a critical regulator of the primed-to-nave transition, with its inhibition consistently enhancing PNT efficiency in high quality across various experimental systems and conditions

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