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When we need energy in a high amount due to increased metabolic demand, such as while doing high-intensity exercise, this stored muscle creatine content in the form of creatine phosphate is converted to ATP, generating a burst of energy we require during these strenuous conditions

2B, C), confirming that cystine restriction elevates oxidative and ferroptosis stress

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And there is evidence showing that SLC5A8 as a Na + -coupled short-chain FA transporter, is downregulated in classical form PTC, which is related to BRAF mutations [107, 108]